Free Radicals and Aging in Parkinsons

Free radical production and their focused on activity on biomolecules have roles in maturing and age-related disorders, for example, Parkinsons disease (PD). There is an age-related increment in oxidative harm to the brain, and aging is viewed as a hazard factor for PD. The normal connection among aging and PD at the cellular level with unique reference to oxidative damage brought about by free radicals. Oxidative harm incorporates mitochondrial dysfunction, dopamine auto-oxidation, α-synuclein conglomeration, glial cell enactment, modifications in calcium flagging, and overabundance free iron.

  • Track 1-1 Neuro Inflammation
  • Track 2-2 Progressive Neurodegenerative Disease
  • Track 3-3 DNA Degeneration
  • Track 4-4 Superoxide radical anion
  • Track 5-5 Dopaminergic neurons

Related Conference of Neuroscience